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Randomised controlled feeding trial, post hoc analysis PubMed records 27 July 2026 as the publication date. Volume 34, issue 9, pages 1732 to 1741.

When every calorie was supplied and matched, a ten hour eating window changed nothing that a sixteen hour one did not

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Journal
Obesity 34(9):1732 to 1741
Authors
Duan D, Carnell S, Pilla SJ, Chung ST, Clark JM, Maruthur NM
Institution
the Division of Endocrinology, Diabetes and Metabolism at Johns Hopkins University School of Medicine
Published
27 July 2026
Source
PMID 42508996 · DOI 10.1002/oby.70270
Design
Post hoc analysis of a twelve week randomised isocaloric and eucaloric feeding trial comparing a ten hour eating window with a sixteen hour usual eating pattern. Registered as NCT03527368. Within and between group differences tested with Wilcoxon signed rank and Mann Whitney U tests.
Sample
Thirty nine adults with obesity and either prediabetes or diet controlled type 2 diabetes.

What isocaloric eucaloric feeding is

Isocaloric means both arms received the same number of calories. Eucaloric means that number was set to hold body weight steady rather than to create a deficit. A feeding study supplies the food rather than asking people to follow instructions. Together they strip out the thing that makes ordinary fasting trials hard to read, which is that people on a short window usually eat less without meaning to.

Drawn from standard trial design vocabulary, not from this paper.

Why they ran it

If a compressed eating window does something beyond making people eat less, then supplying identical calories to both arms should reveal it. The parent trial did that for twelve weeks in adults with obesity and either prediabetes or diet controlled type 2 diabetes, and this analysis asks what was left once intake was held equal.

Drawn from the paper's stated aim.

Thirty nine participants analysed, nineteen on the ten hour window and twenty on the usual sixteen hour pattern. Mean age sixty, mean body mass index 35.2.

Weight loss was 2.7 percent in the time restricted arm and 2.5 percent in the usual pattern arm.

No statistically significant between group differences in eating behaviour, appetite hormones, or inflammatory markers. The panel covered leptin, adiponectin, C reactive protein, cortisol and soluble RAGE, with ghrelin measured across a seventy five gram oral glucose tolerance test.

Both arms increased dietary restraint and decreased disinhibition and hunger on the three factor eating questionnaire.

The authors conclude that both patterns improved eating behaviour in the context of modest and equivalent weight loss.

The numbers

Weight loss, time restricted arm2.7 percent
Weight loss, usual eating pattern arm2.5 percent
Between group differences in eating behaviour, appetite hormones and inflammatory markersnone statistically significant
Eating windows compared10 hours against 16 hours
Mean age and body mass index60 years, body mass index 35.2
Duration and sample12 weeks, 39 participants analysed

Why this might happen

Proposed by the authors This is the explanation the authors offer in their discussion. This study did not test it.

The authors expected a compressed window to change appetite regulation and low grade inflammation independently of energy intake, and they measured the panel that would show it. Nothing separated. Their own reading is that both patterns improved eating behaviour in the context of modest and equivalent weight loss, which places the effect in the weight change rather than in the schedule.

Drawn from the paper's stated aim and conclusion.

What this does not show

  • It does not show a short eating window is useless. It shows that in this trial the benefits ran through the same modest weight loss the control arm achieved, which is a claim about the route rather than about the destination.
  • It cannot rule out a small effect. Thirty nine people split across two arms cannot exclude a difference smaller than the study was sized to find. The authors ask for larger prospective studies for exactly that reason.
  • This is not the trial's primary outcome paper. It is a post hoc analysis of appetite and inflammation measures from a trial designed around other endpoints.
  • It does not describe free living fasting. The food was supplied. That is the point of the design and it is also its limit, because nobody outside a trial eats this way.

Where this leaves us

Hold the calories constant and supply the food, and a ten hour window and a sixteen hour one produce the same weight change and the same hormones.

Thirty nine adults around sixty years old with obesity and prediabetes or early type 2 diabetes, over twelve weeks with all food provided.

A larger feeding trial, which the authors ask for, or the same design in people who are not already metabolically unwell.

Caveats worth holding

  • Post hoc analysis, not the trial's primary report.
  • Nineteen people in one arm and twenty in the other is small for hormone panels.
  • Fasting blood samples at two time points, with ghrelin the only measure taken across a challenge.

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